Simply Stop Model!
Precision Immune Defense Against Sars-CoV-2 COVID19
2020 John A. Catanzaro
Halt Viral Replication Model!
COVID19 identifies 3 with additional 5 possible new strains, a direct result of SMART mutations.
These mutations produced by overproduction of nonstructural proteins on alphavirus plus-strand and minus-strand RNA synthesis. Since the synthesis of SARS-CoV-2 RNA requires ongoing viral protein production, one could speculate whether de-ubiquitinylation by SARS-CoV-2 PLpro protects replicase subunits against proteasomal degradation.
mRNA-1273 can be producing an additional catalog / viral fragment production warehouse, using the same replication model as the virus itself, fueling the ongoing protein intelligence requirements for Next-Gen mutation by their (mRNA-1273) viral-like protein mimicry. Viral / host sustained immune defense, eradication and replication must not use the same COVID19 human viral / host generating replication real time copying / mutation mechanisms.
Real time exponential activated COVID19 in the infected population for sustained immune defense / regulation/ memory must be precision-based with identified susceptibility.
Thus, SMART elements of the COVID19 predicted in a therapeutic treatment model not vaccine. SMART viral copying by proteins produced by mRNA-1273 is ludicrous. PBIMA eradication design for the infected population is a must.
Simply
The virus infects cells that can mutate and uses the human cell as a living host to reproduce itself. The mRNA-1273 uses the same mechanisms as the virus by producing random viral like proteins produced the same way the virus reproduces itself with random proteins that can be used by the virus to develop a higher mutation strain. This can cause reinfection and reactivation. as well as, unwanted autoimmune downstream problems as adverse reactions over time!
